Rehab Centers: Serum Lycopene Decreases the Risk of Stroke in Men: A Population-Based Follow-Up Study.
Serum lycopene decreases the risk of stroke in men: A population-based follow-up study.
Filed under: Rehab Centers
Neurology. 2012 Oct 9; 79(15): 1540-7
Karppi J, Laukkanen JA, Sivenius J, Ronkainen K, Kurl S
Intake of fruits and vegetables and levels of serum carotenoids have been associated with decreased risk of stroke, but the results have been inconsistent. The aim of the present study was to examine whether serum concentrations of major carotenoids, ?-tocopherol and retinol, are related to any stroke and ischemic stroke in men.The study population consisted of 1,031 Finnish men aged 46-65 years in the Kuopio Ischaemic Heart Disease Risk Factor cohort. Serum concentrations of carotenoids retinol and ?-tocopherol were measured by high-performance liquid chromatography. The association between the serum concentrations of lycopene ?-carotene, ?-carotene, ?-tocopherol, and retinol and the risk of strokes was studied by using Cox proportional hazards models.A total of 67 strokes occurred, and 50 of these were ischemic strokes during a median of 12.1 follow-up years. After adjustment for age, examination year, BMI, systolic blood pressure, smoking, serum low-density lipoprotein cholesterol, diabetes, and history of stroke, men in the highest quartile of serum lycopene concentrations had 59% and 55% lower risks of ischemic stroke and any stroke, compared with men in the lowest quartile (hazard ratio [HR] = 0.45, 95% confidence interval [CI] 0.25-0.95, p = 0.036 for any stroke and HR = 0.41; 95% CI 0.17-0.97, p = 0.042 for ischemic stroke). ?-Carotene, ?-carotene, ?-tocopherol, and retinol were not related to the risk of strokes.This prospective study shows that high serum concentrations of lycopene, as a marker of intake of tomatoes and tomato-based products, decrease the risk of any stroke and ischemic stroke in men.
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Effect of vibration stimulation on dysbasia of spastic paraplegia in neuromyelitis optica: a possible example of neuronal plasticity.
Filed under: Rehab Centers
BMJ Case Rep. 2012; 2012:
Lin HN, Nagaoka M, Hayashi Y, Hatori K
We analysed the effect of vibration stimulation (VS) on dysbasia of neuromyelitis optica (NMO). The patient was a 36-year-old woman who was diagnosed with NMO and had difficulties in walking. VS was applied to the lower limb muscles on the left, more spastic, side with an ordinary vibrator. The performance of standing up and walking improved with VS. Even with improved performance after VS, the amount of surface EMG of the lower limbs did not increase as a whole, but the EMG patterns among the lower leg muscles changed remarkably. VS produced reciprocity within antagonistic muscles. Variability of EMG amplitudes decreased remarkably during the walking cycle, not only on the vibrated side, but also on the non-vibrated side. The effect lasted longer than several dozen minutes after the cessation of VS. We conjectured that central pattern generator (CPG) and neuronal plasticity were the result of VS.
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Somatotopic mismatch following stroke: a pathophysiological condition escaping detection.
Filed under: Rehab Centers
BMJ Case Rep. 2012; 2012:
Birznieks I, Logina I, Wasner G
Clinical evaluation of somatosensory deficits in stroke patients is very limited and usually does not include testing of somatotopic organisation, which is a prerequisite for meaningful interpretation of sensory input and sensorimotor control. Detailed tactile testing of the left hand of a 54-year-old patient suffering from sensory deficit and central pain after a right-sided stroke revealed severe distortion of somatotopic sensory maps as evidenced by incorrect localisation of the point stimuli. Unlike previously reported gross somatotopic remapping taking place within reduced representational space after lesion, this is the first case report revealing chaotic scrambled somatosensory maps. While the incidence of such scrambled somatotopic representation of tactile input is not yet known in stroke patients, current observations indicate that in-depth investigations of somatotopic organisation of affected area may reveal the underlying cause for various functional deficits including central pain. Thus, new rehabilitation strategies may need to be developed specifically for such patients.
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Watershed infarcts in a patient after carotid endarterectomy.
Filed under: Rehab Centers
BMJ Case Rep. 2012; 2012:
Hashmi I, Shahzadi F
We report a case that developed watershed infarcts after undergoing carotid endarterectomy. Our patient had been having episodes of transient ischaemic attacks before admission. Carotid Doppler scan before admission showed severe unilateral carotid stenosis on left side. She underwent a carotid endarterectomy procedure. Her continuous blood pressure monitoring stayed normal during and after the procedure. One and half hour after the procedure, she developed right arm weakness. CT scan later revealed watershed infarcts in left cerebral hemisphere. The pathogenesis of watershed infarcts remain debated despite haemodynamic and embolic mechanisms being postulated.
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